Okayama University科研人員發(fā)現(xiàn),用于治療 2 型糖尿病的二甲雙胍罢荡,通過涉及 CD8TIL 線粒體中活性氧的產(chǎn)生和糖酵解增加的機制对扶,誘導(dǎo)腫瘤靶向 CD8+ T 淋巴細胞 (CD8TIL) 的激活和增殖区赵。
Credit: Heiichiro Udono, Okayama University
More information:Yoshihito Tanaka et al, Cutaneous ischemia‐reperfusion injury is exacerbated by IL‐36 receptor antagonist deficiency,Journal of the European Academy of Dermatology and Venereology(2021).
http://dx.doi.org/10.1111/jdv.17767
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